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Nutritional Management of Equine Diseases and Special Cases

Stručný popis

Publikace nabízí stručný a srozumitelný text pro veterináře specializující se na koně s dotazy týkajícími se běžně se vyskytujících nutričních problémů.
Pomáhá veterinářům s podporou koňských pacientů se speciálními nutričními potřebami
Zaměřuje se na nutriční problémy a jejich dopad na různé tělesné systémy
Zahrnuje poníky, miniaturní koně, tažné koně, osly a muly
Nabízí kompletní pokrytí běžných onemocnění a problémů, kterými je léčena výživa
Obsahuje užitečné kapitoly o jedovatých rostlinách a mykotoxinech

Autor: Bryan M. Waldridge (Editor)

Cena s DPH: asi 2776 Kč

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Nakladatel	Wiley-Blackwell
ISBN	9781119191872
Vydání	2017
Vazba	brožovaná
Počet stran	216

Popis

Nutritional Management of Equine Diseases and Special Cases offers a concise, easy-to-comprehend text for equine veterinarians with questions about commonly encountered nutritional problems.
Assists veterinarians in supporting equine patients with special nutritional needs
Focuses on nutritional problems and impact on different body systems
Covers ponies, miniature horses, draft horses, donkeys, and mules
Offers complete coverage of common diseases and problems helped by nutrition
Includes useful chapters on poisonous plants and mycotoxins
About the Author
The editor
Bryan M. Waldridge, DVM, MS, Diplomate, American Board of Veterinary Practitioners (Equine Practice), Diplomate, American College of Veterinary Internal Medicine, is the internist at Park Equine Hospital at Woodford in Versailles, Kentucky, USA. Dr. Waldridge also serves as the resident veterinarian at Old Friends Thoroughbred Retirement Farm in Georgetown, Kentucky, USA.
Reviews
"This is a long overdue addition to the veterinary literature. It helps to demystify the nutritional implications of many commonly seen diseases in a practical, useful, and easily implemented manner. There is no comparable book in the field with this comprehensive information. For even experienced equine practitioners, it will be a useful addition to the library as a resource for determining dietary recommendations for clients." (Doody Enterprises 02/06/2017)

Obsah

Contributors	xiii
Preface	xv
1 Miniature Horses and Ponies	1
1.1 Miniature Horses	1
1.2 General Feeding of Miniature Horses	1
1.3 Pony Feeding	2
References	2
2 Draft Horses Mules and Donkeys	5
2.1 Draft Horses	5
2.2 Donkeys	6
2.3 Mules	7
References	7
3 Gastrointestinal System	9
3.1 The Association between Nutrition and Colic	9
3.1.1 Feeds and Colic: Pastures	9
3.1.2 Feeds and Colic: Dried Forages	10
3.1.3 Feeds and Colic: Concentrates	11
3.1.4 General Practices to Prevent Colic	11
3.2 Nutritional Plans for Horses with Colic	12
3.2.1 Identifying Nutritional Status	12
3.2.2 Nutritional Requirements of Horses with Colic	13
3.3 Routes for Feeding Horses Recovering from Colic	15
3.3.1 Voluntary Intake	15
3.3.2 Supportive Enteral Nutrition	17
3.3.3 Parenteral Nutrition	21
3.4 Diets for Specific Diseases	27
3.4.1 Uncomplicated Colic	27
3.4.2 Equine Gastric Ulcer Syndrome	28
3.4.3 Duodenitis/Proximal Jejunitis	29
3.4.4 Small Intestinal Strangulation	30
3.4.5 Ileal Impaction (Nonstrangulating Small Intestinal Obstruction)	31
3.4.6 Ascending (Large) Colon Impactions	32
3.4.7 Sand Impactions	34
3.4.8 Enteroliths and Fecaliths	35
3.4.9 Ascending Colon Displacement	36
3.4.10 Ascending Colon Volvulus (Large Colon Torsion)	37
3.4.11 Cecal Impactions	39
3.4.12 Cecocecal and Cecocolic Intussusception	39
3.4.13 Descending (Small) Colon Obstructions	40
3.4.14 Descending (Small) Colon Strangulations	41
References	41
4 Muscular System	51
4.1 Myopathies Associated with Nutritional Deficiencies	51
4.1.1 Nutritional Myodegeneration due to Selenium Deficiency	51
4.1.2 Equine Motor Neuron Disease and Vitamin E Deficiency	52
4.1.3 Vitamin E Deficient Myopathy	53
4.1.4 Sporadic Exertional Rhabdomyolysis	54
4.2 Nutrigenomics	55
4.2.1 Chronic Forms of Exertional Rhabdomyolysis	55
4.2.2 Polysaccharide Storage Myopathy	59
4.2.3 Hyperkalemic Periodic Paralysis	66
References	68
5 Endocrine System	73
5.1 Equine Metabolic Syndrome	73
5.1.1 Definition of Equine Metabolic Syndrome	73
5.1.2 Epidemiology	73
5.1.3 Species Age and Sex Predisposition	73
5.1.4 Genetics and Breed Predisposition	73
5.1.5 Risk Factors	74
5.1.6 Geography and Seasonality	74
5.1.7 Associated Conditions and Disorders	74
5.1.8 Clinical Presentation	74
5.1.9 Diagnosis	80
5.1.10 Treatment	82
5.1.11 Possible Complications of Treatment or of the Disease Process	85
5.1.12 Recommended Monitoring	85
5.1.13 Prognosis and Outcome	85
5.1.14 Prevention	85
5.2 Feeding Horses with Pituitary Pars Intermedia Dysfunction	86
5.2.1 Horses with Pituitary Pars Intermedia Dysfunction and Adequate Body Condition	86
5.2.2 Obese Horses with Pituitary Pars Intermedia Dysfunction	87
5.2.3 Horses with Pituitary Pars Intermedia Dysfunction and Thin Body Condition or Horses with PPID that are in Work	87
5.3 Pearls and Considerations	87
5.3.1 Client Education	87
5.3.2 Veterinary Technician Tips	88
References	88
6 Respiratory System	91
6.1 Effects of Inhaled Dust and Potential Aeroallergens on Equine Respiratory Disease	91
6.2 Respirable Dust Deposition in the Airways	91
6.3 Effects of Soaking Hay	93
6.4 Effects of Steam Treating Hay	93
6.5 Feeding Forage Alternatives	93
6.5.1 Haylage	93
6.5.2 Hay Cubes	93
6.5.3 Pellets	95
6.6 Exercise‐Induced Pulmonary Hemorrhage	95
6.7 Acute Interstitial Pneumonia	95
References	95
7 Neurologic System	97
7.1 Cervical Vertebral Malformation	97
7.2 Botulism	98
7.3 Ryegrass Staggers	99
7.4 Equine Degenerative Myelopathy and Neuroaxonal Dystrophy	99
7.5 Equine Motor Neuron Disease	100
7.6 Effect of Form and Dose of Vitamin E on Serum and Cerebrospinal Fluid Concentrations	101
References	102
8 Mycotoxins	103
8.1 Aflatoxins	103
8.1.1 Toxicokinetics	104
8.1.2 Mechanism of Action	104
8.1.3 Toxicity and Clinical Signs	105
8.1.4 Reproductive and Developmental Effects	105
8.1.5 Treatment	106
8.2 Fumonisins	106
8.2.1 Toxicokinetics	107
8.2.2 Mechanism of Action	107
8.2.3 Toxicity and Clinical Signs	107
8.2.4 Treatment	108
8.3 Slaframine	108
8.3.1 Mechanism of Action	109
8.3.2 Toxicity and Clinical Signs	109
8.3.3 Treatment	109
8.4 Trichothecenes	110
8.4.1 Toxicokinetics	110
8.5 Mechanism of Action	111
8.5.1 Toxicity and Clinical Signs	111
8.5.2 Treatment	112
8.6 Zearalenone	112
8.6.1 Toxicokinetics	113
8.6.2 Mechanism of Action	113
8.6.3 Toxicity and Clinical Signs	113
8.7 Treatment	114
8.8 Concluding Remarks	114
Acknowledgment	114
References	114
9 Poisonous Plants	119
9.1 Excessive Salivation Induced by Plants	119
9.2 Colic and Diarrhea‐Inducing Plants	121
9.2.1 Horse Chestnut or Buckeye	121
9.2.2 Field Bindweed (Morning Glory)	122
9.2.3 Oak	123
9.2.4 Mountain Laurel	124
9.2.5 Pokeweed	125
9.2.6 Buttercups	126
9.2.7 Castor Oil Plant	126
9.2.8 Jimson Weed Potato and Tomato	128
9.2.9 Kentucky Coffee Tree	129
9.3 Photodermatitis‐Inducing Plants	129
9.3.1 Primary Photosensitization	129
9.3.2 Secondary Photosensitization	131
9.3.3 Liver Disease‐Inducing Plants	131
9.4 Neurologic Disease‐Inducing Plants	138
9.4.1 Sagebrush	139
9.4.2 Locoweeds and Milkvetches	140
9.4.3 Milkvetch Neurotoxicosis	143
9.4.4 Yellow Star Thistle and Russian Knapweed	143
9.4.5 Horsetail	145
9.4.6 White Snakeroot and Crofton Jimmy or Burrow Weeds	145
9.4.7 Bracken Fern	146
9.4.8 Johnsongrass and Sudangrass	147
9.5 Lameness and Muscle Weakness‐Inducing Plants	149
9.5.1 Black Walnut	149
9.5.2 Hoary Alyssum	150
9.5.3 Coffee Weed or Coffee Senna	150
9.6 Plant‐Induced Calcinosis	151
9.6.1 Day‐Blooming Jessamine	152
9.6.2 Flatweed	153
9.7 Selenium Toxicosis	153
9.7.1 Causes of Selenium Toxicosis	154
9.7.2 Two‐Grooved Milkvetch (Astragalus bisulcatus)	155
9.7.3 False Golden Weed (Oonopsis species)	155
9.7.4 Woody Aster (Xylorhiza glabriuscula)	155
9.7.5 Prince’s Plume (Stanleya pinnata)	155
9.7.6 White Prairie Aster (Aster falcatus)	155
9.7.7 Broom Turpentine Snake or Match Weed (Gutierrezia sarothrae)	156
9.7.8 Gumweed or resinweed (Grindelia spp.)	157
9.7.9 Saltbush (Atriplex spp.)	157
9.7.10 Indian Paintbrush (Castilleja spp.)	157
9.7.11 Beard Tongue (Penstemon spp.)	157
9.7.12 Effects of Acute Selenium Toxicosis	158
9.7.13 Effects of Chronic Selenium Toxicosis	159
9.7.14 Diagnosis of Selenium Toxicosis	160
9.8 Anemia‐Inducing Plants	161
9.8.1 Onions	162
9.8.2 Red Maple	162
9.8.3 Spoiled Sweet Clover	163
9.9 Teratogenic Plants	164
9.10 Sudden Death‐Inducing Plants	165
9.10.1 Cyanide‐Induced Sudden Death	166
9.10.2 Toxicity of Cyanogenic Glycosides	167
9.10.3 Serviceberry or Saskatoon berry (Amelanchier alnifolia)	167
9.10.4 Wild Blue Flax (Linum spp.)	167
9.10.5 Western Chokecherry (Prunus virginiana)	167
9.10.6 Elderberry (Sambuccus spp.)	168
9.10.7 Sorghum Grasses	168
9.10.8 Arrow grass or goose grass (Triglochin spp.)	169
9.10.9 Clinical Effects and Diagnosis of Acute Cyanide Poisoning	169
9.10.10 Treatment of Acute Cyanide Poisoning	170
9.10.11 Cardiac Glycoside‐Induced Sudden Death	170
9.11 Larkspur	175
9.12 Monkshood	176
9.13 Poison Hemlock	176
9.14 Water Hemlock	177
9.15 Yew	178
9.16 Death Camas	179
9.17 Avocado	180
Glossary	180
Supplemental Reading	181
References	182
Index	189